Contents
- 1 Skin in atopic dermatitis is deficient in
- 2 The infectious agent most often involved in Atopic Dermatitis is
- 3 Severe AD in infancy is a major risk factor for
- 4 Not true regarding atopic dermatitis in children
- 5 Rash identical to that of AD is seen in
- 6 In short
- 7 Atopy patch testing
- 8 Treatment
- 9 First line therapy
- 10 Local side effects of long-term topical corticosteroid
Skin in atopic dermatitis is deficient in
A. Elastin
B. Glutamine
C. Ceramides
D. Cysteine
The infectious agent most often involved in Atopic Dermatitis is
A. Propionibacterium acnes
B. Streptococcus pyogenes
C. Coryneform bacteria
D. Staphylococcus aureus
Severe AD in infancy is a major risk factor for
A. Food allergies
B. Asthma
C. Allergic rhinitis
D. Acne
Not true regarding atopic dermatitis in children
A. The diaper area is usually affected in infants
B. Visible flexural dermatitis in older children
C. Continues throughout the day and worsens at night
D. Eczema of cheeks, forehead seen in children under 4 years
Rash identical to that of AD is seen in
A. Netherton syndrome
B. Asteatotic eczema
C. Wiskott-Aldrich syndrome
D. Nummular dermatitis
In short
Atopy patch testing
Atopy patch testing is still considered investigational in patients with AD because there are no standardized methods of application or test interpretation.
Treatment
The treatment of AD should be directed at restoring the skin barrier, which includes hydrating and repairing the skin, limiting itching, and decreasing inflammation when necessary.
First line therapy
First line therapy of AD is anti-inflammatory treatment with topical corticosteroids.
Local side effects of long-term topical corticosteroid
Possible local side effects of long-term topical corticosteroid use include
- Striae – stretch marks
- Petechiae – small red/purple spots
- Telangiectasia – small, dilated blood vessels on the surface of the skin
- Skin thinning
- Atrophy
- Acne.
